Visceral, hematologic and bacteriologic changes and neurologic outcome after cardiac arrest in dogs. The visceral post-resuscitation syndrome
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2019, Kidney InternationalCitation Excerpt :One reason for this belief was that the leading causes of AKI (e.g., sepsis, major surgery, heart failure, and hypovolemia) are all associated with hypoperfusion and shock,1 and ischemic injury can cause extensive cell death (e.g., acute tubular necrosis). However, it is becoming increasingly clear that ischemia-reperfusion injury is not the only mechanism of S-AKI, but rather multiple mechanisms must be at play.38–40 For instance, S-AKI may develop in the absence of renal hypoperfusion and clinical signs of hemodynamic instability21,41,42 and in the presence of normal or increased global renal blood flow.41,43–49
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